Chú thích Ứng kích ôxi hóa

  1. ↑ de Diego-Otero Y, Romero-Zerbo Y, el Bekay R, Decara J, Sanchez L, Rodriguez-de Fonseca F, del Arco-Herrera I. (tháng 3 năm 2009). “Alpha-tocopherol protects against oxidative stress in the fragile X knockout mouse: an experimental therapeutic approach for the Fmr1 deficiency.”. Neuropsychopharmacology 34 (4): 1011–26. PMID 18843266. doi:10.1038/npp.2008.152.  Chú thích sử dụng tham số |month= bị phản đối (trợ giúp)
  2. ↑ Gems D, Partridge L (tháng 3 năm 2008). “Stress-response hormesis and aging: "that which does not kill us makes us stronger"” (PDF). Cell Metab. 7 (3): 200–3. PMID 18316025. doi:10.1016/j.cmet.2008.01.001.  Chú thích sử dụng tham số |month= bị phản đối (trợ giúp)
  3. ↑ Schafer FQ, Buettner GR (2001). “Redox environment of the cell as viewed through the redox state of the glutathione disulfide/glutathione couple”. Free Radic. Biol. Med. 30 (11): 1191–212. PMID 11368918. doi:10.1016/S0891-5849(01)00480-4. 
  4. ↑ Lennon SV, Martin SJ, Cotter TG (1991). “Dose-dependent induction of apoptosis in human tumour cell lines by widely diverging stimuli”. Cell Prolif. 24 (2): 203–14. PMID 2009322. doi:10.1111/j.1365-2184.1991.tb01150.x. 
  5. ↑ Valko M, Morris H, Cronin MT (tháng 5 năm 2005). “Metals, toxicity and oxidative stress”. Curr. Med. Chem. 12 (10): 1161–208. PMID 15892631. doi:10.2174/0929867053764635.  Chú thích sử dụng tham số |month= bị phản đối (trợ giúp)
  6. ↑ Evans MD, Cooke MS. Factors contributing to the outcome of oxidative damage to nucleic acids. Bioessays. 2004 May;26(5):533–42. Review. PMID 15112233
  7. ↑ Lelli JL, Becks LL, Dabrowska MI, Hinshaw DB (1998). “ATP converts necrosis to apoptosis in oxidant-injured endothelial cells”. Free Radic. Biol. Med. 25 (6): 694–702. PMID 9801070. doi:10.1016/S0891-5849(98)00107-5. 
  8. ↑ Lee YJ, Shacter E (1999). “Oxidative stress inhibits apoptosis in human lymphoma cells”. J. Biol. Chem. 274 (28): 19792–8. PMID 10391922. doi:10.1074/jbc.274.28.19792. 
  9. ↑ Sies, H. (1985). “Oxidative stress: introductory remarks”. Trong H. Sies, (Ed.). Oxidative Stress. London: Academic Press. tr. 1–7. 
  10. ↑ Docampo, R. (1995). “Antioxidant mechanisms”. Trong J. Marr and M. Müller, (Eds.). Biochemistry and Molecular Biology of Parasites. London: Academic Press. tr. 147–160. 
  11. 1 2 Rice-Evans CA, Gopinathan V (1995). “Oxygen toxicity, free radicals and antioxidants in human disease: biochemical implications in atherosclerosis and the problems of premature neonates”. Essays Biochem. 29: 39–63. PMID 9189713. 
  12. ↑ Seaver LC, Imlay JA (tháng 11 năm 2004). “Are respiratory enzymes the primary sources of intracellular hydrogen peroxide?”. J. Biol. Chem. 279 (47): 48742–50. PMID 15361522. doi:10.1074/jbc.M408754200.  Chú thích sử dụng tham số |month= bị phản đối (trợ giúp)
  13. ↑ Messner KR, Imlay JA (tháng 11 năm 2002). “Mechanism of superoxide and hydrogen peroxide formation by fumarate reductase, succinate dehydrogenase, and aspartate oxidase”. J. Biol. Chem. 277 (45): 42563–71. PMID 12200425. doi:10.1074/jbc.M204958200.  Chú thích sử dụng tham số |month= bị phản đối (trợ giúp)
  14. ↑ Imlay JA (2003). “Pathways of oxidative damage”. Annu. Rev. Microbiol. 57: 395–418. PMID 14527285. doi:10.1146/annurev.micro.57.030502.090938. 
  15. ↑ Nijs J, Meeus M, De Meirleir K (2006). “Chronic musculoskeletal pain in chronic fatigue syndrome: recent developments and therapeutic implications.”. Man Ther 11 (3): 187–91. PMID 16781183. doi:10.1016/j.math.2006.03.008. 
  16. ↑ Meyers DG, Maloley PA, Weeks D (1996). “Safety of antioxidant vitamins”. Arch. Intern. Med. 156 (9): 925–35. PMID 8624173. doi:10.1001/archinte.156.9.925. 
  17. ↑ Ruano-Ravina A, Figueiras A, Freire-Garabal M, Barros-Dios JM (2006). “Antioxidant vitamins and risk of lung cancer”. Curr. Pharm. Des. 12 (5): 599–613. PMID 16472151. doi:10.2174/138161206775474396. 
  18. ↑ Pryor WA (2000). “Vitamin E and heart disease: basic science to clinical intervention trials”. Free Radic. Biol. Med. 28 (1): 141–64. PMID 10656300. doi:10.1016/S0891-5849(99)00224-5. 
  19. ↑ Boothby LA, Doering PL (2005). “Vitamin C and vitamin E for Alzheimer's disease”. Ann Pharmacother 39 (12): 2073–80. PMID 16227450. doi:10.1345/aph.1E495. 
  20. ↑ Kontush K, Schekatolina S (2004). “Vitamin E in neurodegenerative disorders: Alzheimer's disease”. Ann. N. Y. Acad. Sci. 1031: 249–62. PMID 15753151. doi:10.1196/annals.1331.025. 
  21. ↑ Fong JJ, Rhoney DH (2006). “NXY-059: review of neuroprotective potential for acute stroke”. Ann Pharmacother 40 (3): 461–71. PMID 16507608. doi:10.1345/aph.1E636. 
  22. ↑ Larsen PL (1993). “Aging and resistance to oxidative damage in Caenorhabditis elegans”. Proc. Natl. Acad. Sci. U.S.A. 90 (19): 8905–9. PMC 47469. PMID 8415630. doi:10.1073/pnas.90.19.8905. 
  23. ↑ Helfand SL, Rogina B (2003). “Genetics of aging in the fruit fly, Drosophila melanogaster”. Annu. Rev. Genet. 37: 329–48. PMID 14616064. doi:10.1146/annurev.genet.37.040103.095211. 
  24. ↑ Publication demonstrating that oxidative stress is promoting life span
  25. ↑ Tapia PC (2006). “Sublethal mitochondrial stress with an attendant stoichiometric augmentation of reactive oxygen species may precipitate many of the beneficial alterations in cellular physiology produced by caloric restriction, intermittent fasting, exercise and dietary phytonutrients: "Mitohormesis" for health and vitality”. Med. Hypotheses 66 (4): 832–43. PMID 16242247. doi:10.1016/j.mehy.2005.09.009. 
  26. ↑ Sohal RS, Mockett RJ, Orr WC (2002). “Mechanisms of aging: an appraisal of the oxidative stress hypothesis”. Free Radic. Biol. Med. 33 (5): 575–86. PMID 12208343. doi:10.1016/S0891-5849(02)00886-9. 
  27. ↑ Sohal RS (2002). “Role of oxidative stress and protein oxidation in the aging process”. Free Radic. Biol. Med. 33 (1): 37–44. PMID 12086680. doi:10.1016/S0891-5849(02)00856-0. 
  28. ↑ Rattan SI (2006). “Theories of biological aging: genes, proteins, and free radicals”. Free Radic. Res. 40 (12): 1230–8. PMID 17090411. doi:10.1080/10715760600911303. 
  29. ↑ Bjelakovic G, Nikolova D, Gluud LL, Simonetti RG, Gluud C (2007). “Mortality in randomized trials of antioxidant supplements for primary and secondary prevention: systematic review and meta-analysis”. JAMA 297 (8): 842–57. PMID 17327526. doi:10.1001/jama.297.8.842. . See also the letter to JAMA by Philip Taylor và Sanford Dawsey and the reply by the authors of the original paper.
  30. ↑ Devasagayam, TPA; Tilac JC, Boloor KK, Sane Ketaki S, Ghaskadbi Saroj S, Lele RD (tháng 10 năm 2004). “Free Radicals and Antioxidants in Human Health: Current Status and Future Prospects”. Journal of Association of Physicians of India 52: 796.  Chú thích sử dụng tham số |coauthors= bị phản đối (trợ giúp); Kiểm tra giá trị ngày tháng trong: |accessdate= (trợ giúp); ||ngày truy cập= cần |url= (trợ giúp)
  31. ↑ Nathan C, Shiloh MU (2000). “Reactive oxygen and nitrogen intermediates in the relationship between mammalian hosts and microbial pathogens”. Proc. Natl. Acad. Sci. U.S.A. 97 (16): 8841–8. PMC 34021. PMID 10922044. doi:10.1073/pnas.97.16.8841.